In botulinum toxin poisoning, acetylcholine levels in the synaptic cleft are:, A. Increased due to reduced breakdown, B. Normal but ineffective at receptors, C. Decreased due to impaired release, D. Unchanged due to receptor blockade, Botulinum toxin most likely results in:, A. Sustained muscle contraction due to excess acetylcholine, B. Spastic paralysis caused by receptor overstimulation, C. Flaccid paralysis due to failure of neuromuscular transmission, D. Initial muscle twitching followed by rigidity, In a patient with botulinum poisoning, which step of neuromuscular junction physiology remains intact?, A. Propagation of action potential along motor neuron, B. Calcium-dependent vesicle fusion, C. Exocytosis of neurotransmitter, D. Depolarization of motor end plate, Failure of neuromuscular transmission in botulinum poisoning is primarily due to disruption of:, A. Postsynaptic ligand-gated sodium channels, B. Presynaptic vesicular exocytosis of acetylcholine, C. Acetylcholinesterase activity in the synaptic cleft, D. Voltage-gated sodium channels in muscle membrane.

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